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偏最小平方法的結構方程模型(PLS-SEM)：應用SmartPLS

為了解決Model Y vs Model 3 P的問題，作者張紹勳 這樣論述：

　　⊙從概念、原理，深入淺出地向讀者介紹PLS的常用模型與應用。 　　⊙理論與實務兼具，以實例展示SmartPLS分析過程，學以致用。 　　⊙適合社會科學、生醫、工程、財經等研究領域使用。 　　隨書附贈光碟含資料檔、專案檔、模型檔。 　　結構方程模型(structural equation model, SEM)結合了傳統統計學上的因素分析與路徑分析技術，已成為當前發表文章中常見的統計分析。SmartPLS具有可分析小樣本、能精確估計中介和干擾等問題的特性，幫助研究者自動、快速完成統計程式，因此廣受資管、行銷、商學、運動休閒、健康、旅遊等領域的愛載，迄今逐漸成為社會科學及

生醫的主流分析軟體。 　　本書以軟體SmartPLS為分析工具，從概念、原理到實作，一步一步向讀者介紹PLS的常用模型與應用上需注意的問題，並以實例展示SmartPLS分析過程，適合研讀領域有：社會科學、運輸、農業、生物醫學、藥學、製藥、電腦科學、工程、能源、技術、環境科學、材料科學、管理、會計、心理學、商學、經濟、計量經濟、財務等。  

Model Y vs Model 3 P進入發燒排行的影片

使用理財機器人的行為意圖之研究

為了解決Model Y vs Model 3 P的問題，作者林昱德 這樣論述：

本研究以探討使用者使用理財機器人之使用行為相關研究，目的為探討使用者使用因素，提供未來後續業界之參考，以及找出現階段理財機器人使用者的描述性統計分析。本研究以有使用過銀行推出之理財機器人作為研究對象，於 2022年 7月 14日於網路進行正式問卷投放，回收後進行資料分析，經過問卷後台揭露，本次問卷研究投放人數為 4765 人，回收 490 份問卷，有效得 387份，有效回收率為 78.79%。研究架構以 UTAUT2 為基礎，並加入感知風險成為新的會影響使用意圖的因素。研究結果顯示，績效預期、社群影響、促進條件、價格價值以及習慣會對行為意圖產生顯著正向影響；努力預期以及感知風險對行為意圖則是

沒有影響；行為意圖以及習慣對使用行為有顯著正向影響；促進條件對使用意圖則無影響。希冀本研究可以作為相關單位的參考依據。

多變量統計之線性代數基礎：應用SPSS分析

為了解決Model Y vs Model 3 P的問題，作者張紹勳,林秀娟 這樣論述：

　　在自然與社會科學領域，隨著研究方法的複雜化，應用多變量統計方法來分析資料的機會也相對增加。近年來，研究生人數逐年增加，基於學位論文撰寫的需要，多變量統計方法及SPSS等套裝統計軟體的運用成為不可或缺的能力！ 　　本書介紹的多變量分析內容，包含平均數之假設檢定、多變量變異數分析、多元迴歸分析、典型相關分析、區別分析、主成分分析、因素分析、集群分析和多元標度法。透過統計軟體SPSS探討，結合理論、方法與統計從基礎統計知識引導，並輔以練習題與範例，讓學習者能做中學，以增進學習效果。 本書特色 　　◎本書完整介紹多變量分析於SPSS軟體上的操作與統計應用。 　　◎從多變量

分析基礎教起，架構明確搭配範例練習，結合理論與應用性。 　　◎使用SPSS V25介面操作，從使用者角度整理編排，讓研究過程更輕鬆。 　　◎適用於生物學、經濟學、市場行銷、工程學、遺傳學、醫學、教育學、心理學、 　　社會科學、生產管理、風險管理、人資管理、航運管理、財務金融、會計和公共衛生等學術領域。 　　隨書附贈資料檔光碟  

An Integrated Approach For Uncovering Novel DNA Methylation Biomarkers For Non-small Cell Lung Carcinoma

為了解決Model Y vs Model 3 P的問題，作者VAIBHAV KUMAR SUNKARIA 這樣論述：

Introduction - Lung cancer is one of primal and ubiquitous cause of cancer related fatalities in the world. Leading cause of these fatalities is non-small cell lung cancer (NSCLC) with a proportion of 85%. The major subtypes of NSCLC are Lung Adenocarcinoma (LUAD) and Lung Small Cell Carcinoma (LUS

C). Early-stage surgical detection and removal of tumor offers a favorable prognosis and better survival rates. However, a major portion of 75% subjects have stage III/IV at the time of diagnosis and despite advanced major developments in oncology survival rates remain poor. Carcinogens produce wide

spread DNA methylation changes within cells. These changes are characterized by globally hyper or hypo methylated regions around CpG islands, many of these changes occur early in tumorigenesis and are highly prevalent across a tumor type.Structure - This research work took advantage of publicly avai

lable methylation profiling resources and relevant comorbidities for lung cancer patients extracted from meta-analysis of scientific review and journal available at PubMed and CNKI search which were combined systematically to explore effective DNA methylation markers for NSCLC. We also tried to iden

tify common CpG loci between Caucasian, Black and Asian racial groups for identifying ubiquitous candidate genes thoroughly. Statistical analysis and GO ontology were also conducted to explore associated novel biomarkers. These novel findings could facilitate design of accurate diagnostic panel for

practical clinical relevance.Methodology - DNA methylation profiles were extracted from TCGA for 418 LUAD and 370 LUSC tissue samples from patients compared with 32 and 42 non-malignant ones respectively. Standard pipeline was conducted to discover significant differentially methylated sites as prim

ary biomarkers. Secondary biomarkers were extracted by incorporating genes associated with comorbidities from meta-analysis of research articles. Concordant candidates were utilized for NSCLC relevant biomarker candidates. Gene ontology annotations were used to calculate gene-pair distance matrix fo

r all candidate biomarkers. Clustering algorithms were utilized to categorize candidate genes into different functional groups using the gene distance matrix. There were 35 CpG loci identified by comparing TCGA training cohort with GEO testing cohort from these functional groups, and 4 gene-based pa

nel was devised after finding highly discriminatory diagnostic panel through combinatorial validation of each functional cluster.Results – To evaluate the gene panel for NSCLC, the methylation levels of SCT(Secritin), FOXD3(Forkhead Box D3), TRIM58(Tripartite Motif Containing 58) and TAC1(Tachikinin

1) were tested. Individually each gene showed significant methylation difference between LUAD and LUSC training cohort. Combined 4-gene panel AUC, sensitivity/specificity were evaluated with 0.9596, 90.43%/100% in LUAD; 0.949, 86.95%/98.21% in LUSC TCGA training cohort; 0.94, 85.92%/97.37 in GEO 66

836; 0.91,89.17%/100% in GEO 83842 smokers; 0.948, 91.67%/100% in GEO83842 non-smokers independent testing cohort. Our study validates SCT, FOXD3, TRIM58 and TAC1 based gene panel has great potential in early recognition of NSCLC undetermined lung nodules. The findings can yield universally accurate

and robust markers facilitating early diagnosis and rapid severity examination.