Z1a的問題，透過圖書和論文來找解法和答案更準確安心。 我們找到下列包括價格和評價等資訊懶人包

Veloce How to Restore Kawasaki Z1, Z/KZ900 & Z/KZ1000: Your Step-by-Step Colour Illustrated Guide to Complete Restoration

為了解決Z1a的問題，作者Rooke, Chris 這樣論述：

Kawasaki Z1, Z/KZ900 & Z/KZ1000 is for all owners and enthusiasts of the legendary Kawasaki Z1 900, Z/KZ900 and Z1000 built between 1972 and 1980. The book covers the complete restoration of a 1976 KZ900, from the sourcing of the bike to its completion as a fully restored machine. Every area

is covered starting with advice on the different models, spares availability, and where best to source a bike to restore. Every area of the restoration is then covered in full detail including the engine, frame, gearbox, wheels, suspension and forks, brakes, ancillaries, bodywork and electrics, acco

mpanied by hundreds of clear color photos to illustrate the text. Additional work on a 1974 Z1A model adds complementary information. What really sets this manual apart, however, is the style in which it is written: not as some dull and distant workshop manual but in a friendly, humorous manner by a

n enthusiast of many years who is able to involve and entertain the reader, as well as guiding them through the restoration process. This is the third restoration guide from Chris Rooke, whose previous works have been a massive hit with mechanics, enthusiasts and restorers throughout the world.

Z1a進入發燒排行的影片

探討斑馬魚Nogo共受體在斑馬魚發育時期對於側線神經丘形成所扮演的角色

為了解決Z1a的問題，作者韓皓偉 這樣論述：

斑馬魚的側線系統是由數個稱為神經丘的感覺器官所構成。一些研究指出斑馬魚的神經丘形成是受到側線神經系統的控制。另一方面，神經細胞表面的兩組Nogo三元接受器複合體：NgR1/LINGO-1/p75NTR以及NgR1/LINGO-1/TROY，會影響神經纖維的外生長與再生。在本次研究中，我們想要探討三個Nogo共受體：p75NTR，LINGO-1以及TROY，在斑馬魚側線神經丘形成上所扮演的角色。我們利用全覆式原位雜交技術來探討三個Nogo共受體的mRNA在斑馬魚胚胎的表現分布情形。結果顯示，斑馬魚p75NTR的mRNA表現在原腎管與三叉神經的位置。此外，p75NTR的mRNA也對稱表現在腦部的

部份區域，目前我們還在進一步研究這是由何種細胞或組織所表現的。而斑馬魚LINGO-1 的mRNA則是專一性的表現在中樞神經的位置。至於斑馬魚TROY的 mRNA在24小時的胚胎中主要表現在中腦的腦室，而在晚期則表現在中腦腦室邊緣、耳囊及前腸。接下來我們利用morpholino反義寡核苷酸(MO)技術來研究三個Nogo共受體在斑馬魚側線神經丘形成所扮演的角色。在經由p75NTR及TROY morpholino反義寡核苷酸注射之後，斑馬魚後側線神經丘的個數於受精後72小時明顯減少，但是LINGO-1的morpholino反義寡核苷酸注射則對於側線發育沒有影響。此外，我們嘗試利用neurogenin

-1-MO來回復由p75NTR-MO及TROY-MO所造成的神經丘缺陷，然而我們的結果顯示，在合打p75NTR-MO (或TROY-MO)和neurogenin-1-MO之後，神經丘的個數依舊是明顯減少。在這次的研究中，為建立整個Nogo系統在斑馬魚側線發育中可能的訊息傳遞路徑，我們也注射了Nogo蛋白及其接受器的morpholino反義寡核苷酸至斑馬魚的胚胎中，結果顯示，Nogo-C2及NgRH1a 的morpholino反義寡核苷酸注射也會造成神經丘個數減少。由這些morpholino反義寡核苷酸注射的結果，我們推測斑馬魚的p75NTR、TROY及NgRH1a可能會形成一種三元的接受器複合

體，並且Nogo-C2會結合到此複合體來影響斑馬魚側線神經丘的形成