走在汽車革命的路上論文書籍站
squamous epithelial 的問題，透過圖書和論文來找解法和答案更準確安心。 我們找到下列包括價格和評價等資訊懶人包
squamous epithelial 的問題，我們搜遍了碩博士論文和台灣出版的書籍，推薦Margret, Dr Jerlin,Dhanapal, Dr Raghu,Kumar, Dr Goma寫的 Epithelial Mesenchymal Transition in Oral Squamous Cell Carcinoma 和Rao, Nagarjun, M.D./ Moran, Cesar A., M.D.的 Pulmonary Pathology都 可以從中找到所需的評價。
另外網站Squamous epithelium (video) | Tissues - Khan Academy也說明：What does the squamous epithelium look like, where is it found, and what are its functions? Let's find out in this video. Created by Moumita Sen ...
這兩本書分別來自 和所出版 。
國立宜蘭大學 生物技術與動物科學系動物科學碩士班 花國鋒所指導 理昱傑的 人蔘皂甙 M1 抑制人類口腔癌之效果及作用機轉 （2021），提出squamous epithelial 關鍵因素是什麼，來自於口腔鱗狀上皮細胞癌、人蔘皂甙、生物轉化、細胞凋亡、遷移、異種移植。
而第二篇論文國防醫學院 生命科學研究所 李曉屏、王正康所指導 廖恆毅的 Matriptase在口腔癌細胞惡性化及藥物治療中扮演角色之研究 （2021），提出因為有 口腔癌、藥物治療的重點而找出了 squamous epithelial 的解答。
最後網站Squamous hyperplasia則補充：Benign thickening of squamous epithelium (more than 15 cell layers) without atypia ... Most common epithelial change associated with keratinizing ...
Epithelial Mesenchymal Transition in Oral Squamous Cell Carcinoma
為了解決squamous epithelial 的問題，作者Margret, Dr Jerlin,Dhanapal, Dr Raghu,Kumar, Dr Goma 這樣論述：
人蔘皂甙 M1 抑制人類口腔癌之效果及作用機轉
為了解決squamous epithelial 的問題，作者理昱傑 這樣論述：
口腔鱗狀上皮細胞癌(Oral squamous cell carcinoma, OSCC)占臺灣所有惡性腫瘤的 5.8%，發病率逐漸上升，為全世界常見的惡性腫瘤，患者存活率極低，因此需要新的有效治療方法來控制口腔鱗狀上皮細胞癌。本篇研究我們製備人蔘皂甙 M1 (20-O--D-glucopyranosyl-20(S)-protopanaxadiol)，為人蔘皂甙主要的去醣基化代謝物，經由真菌 SP-LSL-002 於原料三七葉生物轉化而得，並用於探討人蔘皂甙 M1 於口腔癌細胞及動物模式之抗癌作用及其作用機制。研究結果顯示，人蔘皂甙 M1 可抑制人類口腔癌鱗狀上皮細胞株 SAS 和 OEC
-M1 之存活率。進一步探討人蔘皂甙 M1 之作用機轉，我們發現人蔘皂甙 M1 可增加口腔鱗狀上皮癌細胞之 Bak、Bad 和 p53 蛋白表現，並造成細胞 DNA 斷裂、細胞週期停滯於 G1 期、PI/Annexin V 雙重染色呈現陽性以及 Caspase-3/9 活化，進而促進細胞凋亡。研究結果也證明，人蔘皂甙 M1 可顯著降低 SAS 和 OEC-M1 細胞株之細胞群落生成和遷移能力，並降低癌細胞轉移相關蛋白 Vimentin 之表現。除此之外，以口服或皮下注射給予人蔘皂甙 M1 可明顯抑制 SAS 腫瘤細胞異種移植小鼠之腫瘤生長。綜合以上結果，人蔘皂甙 M1 具有潛力成為口腔癌之治療
藥物。
Pulmonary Pathology
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為了解決squamous epithelial 的問題，作者Rao, Nagarjun, M.D./ Moran, Cesar A., M.D. 這樣論述：
Intended for busy practitioners, Pulmonary Pathology is a pithy, pocket-sized guide to all of the key pulmonology entities and diagnoses that pulmonologists see in daily practice. It addresses non-neoplastic conditions including Infections, Granulomatous Diseases, Acute Lung Injury, Idiopathic Inter
stitial Pneumonias, Vasculitis, Histiocytoses, Lung Pathology in Systemic Diseases, Transplant-Related Lung Pathology, and Miscellaneous Non-Neoplastic Conditions. Neoplasia entities include Benign Epithelial Neoplasia, Pre-Invasive Neoplasia, Squamous Cell Carcinoma, Adenocarcinoma, Neuroendocrine
Neoplasia, Other Epithelial Neoplasia, Mesenchymal Neoplasia, Lymphoproliferative Disorders, Pleural Tumors, Metastatic Tumors, and Tumor-Like Conditions. The book features a bulleted format replete with illustrations for quick information retrieval. It will be a handy summary and quick reference fo
r pulmonary pathology residents and will serve as a portable refresher course for more experienced pathologists. The Demos Surgical Pathology Guides series presents in summary and visual form the basic knowledge base that every practicing pathologist needs each working day. Series volumes cover the
major specialty areas of surgical pathology, and coverage emphasizes the key entities and diagnoses that pathologists will see in practice, and that they must know whether in training or practice. The emphasis is on the basic morphology with newer techniques represented where they are frequently use
d. The series provides a handy summary and quick reference that any pathology resident or fellow will find useful. Experienced practitioners will find the series valuable as a portable refresher course or review tool."
Matriptase在口腔癌細胞惡性化及藥物治療中扮演角色之研究
為了解決squamous epithelial 的問題，作者廖恆毅 這樣論述：
據衛生福利部統計，自民國81年以來，惡性腫瘤已成為台灣第一大死因，口腔癌死亡率在全台灣排名為第六位，並在台灣男性中位列第四位。流行病學調查顯示，大約近九成的口腔癌患者有嚼食檳榔的習慣。檳榔中的主要成分檳榔鹼，它是誘發腫瘤發生和腫瘤進展的主要致癌物質。口腔癌治療目前主要方法為外科手術切除後，或聯合放射治療和化學治療。然而，這些治療方法受多種因素的影響，特別在腫瘤較大或合併轉移的病患上並不完全有效。因此，目前迫切需要進一步了解口腔癌相關機制，以提高治療的效果。Matriptase是膜結合絲氨酸蛋白酶，它與不同類型表皮起源的癌症進展有關，包括口腔粘膜和鱗狀上皮。過去的研究表明，matriptase
的過度表達會導致人類細胞的癌症進展和預後不良。因此本研究將探討matriptase 在口腔癌惡性化及抗藥性中所扮演的角色。本研究使用檳榔鹼反覆刺激培養的人類口腔癌細胞 (OEC-M1)，產生更惡性化的口腔癌細胞。為了評估口腔癌細胞受檳榔致癌物影響的變化，研究應用全自動細胞紀錄系統 (ECIS) 來記錄細胞微運動、增殖、遷移和侵襲。matriptase與惡性化及抗藥性的相關途徑，則透過西方點墨法進行分析比較。另外，使用臨床一線的化療藥物順鉑 (Cisplatin)，以及過去研究中能明顯抑制腫瘤細胞生長的穿心蓮內酯 (Andrographolide)及大麻二酚 (Cannabidiol)，探討m
atriptase在癌症治療中對抗藥性的影響。為了確認matriptase扮演的角色，本研究使用CRISPR/Cas9技術剔除OEC-M1中的matriptase，並與控制組做對照。結果顯示，在口腔癌惡性化實驗中，在經過多次的高濃度檳榔鹼篩選後，OEC-M1的遷移能力跟抗藥性皆有增加，並且matriptase 的表現量有增加的趨勢，在matriptase基因剔除後，口腔癌細胞的增生及遷移能力有明顯的降低。在抗藥性的實驗中，matriptase剔除後，不同藥物對OEC-M1的毒性明顯增加，造成更多的細胞凋亡。在本研究中，matriptase 在癌細胞惡性化和相關化學藥物治療中扮演重要的角色。相信
在未來相關應用的研究可提升癌症化學藥物治療的效果，並降低預後不良的發生。