APM Monaco的問題,透過圖書和論文來找解法和答案更準確安心。 我們找到下列包括價格和評價等資訊懶人包

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國防醫學院 護理研究所 王桂芸所指導 王秀敏的 胸腔重症病人成功脫離呼吸器之預測指標: 以回溯性研究探討 (2021),提出APM Monaco關鍵因素是什麼,來自於胸腔重症、成功脫離呼吸器、預測指標。

而第二篇論文國立臺灣師範大學 生命科學系 趙淑妙所指導 陳章輝的 松科和羅漢松的質體基因組演化和親緣關係與裸子植物中乙酰輔酶A羧化酶基因的演化 (2018),提出因為有 plastome、gymnosperms、Pinaceae、Podocarpaceae、conifers、evolution、plastid、chloroplasts、plastid-to-nucleus gene transfers、accD、acetyl-CoA carboxylase的重點而找出了 APM Monaco的解答。

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胸腔重症病人成功脫離呼吸器之預測指標: 以回溯性研究探討

為了解決APM Monaco的問題,作者王秀敏 這樣論述:

研究背景與動機:近年來全球長期使用呼吸器病人數持續增加,使重症加護病房呼吸器使用需求亦同步增加,造成社會醫療資源龐大支出負擔,縮短呼吸器持續使用時間,可降低呼吸器使用相關併發症、發病率、死亡率及住院費用的風險,協助病人盡早脫離呼吸器實為照護重點;重症加護病房評估病人脫離呼吸器訓練,多為醫師主觀性經驗評估,缺乏有效的科學數據支持,主觀的經驗判斷會導致參差不齊的脫離呼吸器成功率,甚至延遲或脫離失敗,使病人病情加劇變化,故為預防對病人延遲或脫離失敗的判斷,客觀的呼吸器脫離預測指標對於臨床醫療人員評估病人脫離呼吸器的準備至關重要,故綜合國內外文獻探討胸腔重症病人成功脫離呼吸器之相關預測指標。目的:探

討胸腔重症病人成功脫離呼吸器之相關因素與預測指標。研究方法:此為一回溯性病歷相關性研究,於北區某醫學中心內科加護中心之胸腔疾病病人為研究對象,收集時間為民國105年7月1日至110年6月30日,共收案人數418位,以基本屬性(人口學變項、疾病特性)、生理功能參數(中樞神經系統、呼吸系統、心臟血管系統、代謝狀態、腎臟系統)及呼吸器脫離指標為自變項,以呼吸器脫離結果為依變項,使用SPSS 23.0統計方式,包含百分比、平均數、標準差、T檢定、卡方檢定及逐步邏輯斯迴歸,建構胸腔重症病人成功脫離呼吸器之多因素預測模型,以P≦0.05達統計顯著意義。結果:研究對象平均年齡68 ± 18歲,以男性居多65

.3%,呼吸器使用時間平均7.4 ± 8天,人口學變項及疾病特性與成功脫離呼吸器之關係採卡方及獨立T檢定,達統計顯著意義有平均年齡、APACH I、入住加護中心天數、住院天數及矯正查爾森共病指數,生理功能參數及呼吸器脫離指數與成功脫離呼吸器之關係採獨立樣本T檢定,達統計顯著意義有GCS、呼吸次數、潮氣容積、血中氧合濃度、血紅素、血比容、血中酸鹼值、氧氣分壓、碳酸氫鹽、血清白蛋白、白血球、24小時前體液平衡狀態、鉀離子、鈣離子、氧合指數。使用逐步邏輯斯迴歸建立預測模型,三天內預測成功脫離呼吸器之預測因子包含血紅素(勝算比:1.377,p<0.001)、血中酸鹼值(勝算比:0.0001,p=0.0

03)、氧氣分壓(勝算比:1.015,p<0.001)、鉀離子(勝算比:0.36,p=0.002)、24小時前體液平衡狀態(勝算比:1.0,p=0.005),準確度為87.1%,進一步預測成功脫離呼吸器使用概率受試者操作曲線(receiver operating characteristic curve, ROC curve)為0.906。結論:胸腔重症病人三天內預測成功脫離呼吸器5個指標,分別為血紅素、血中酸鹼值、氧氣分壓、鉀離子、24小時前體液平衡狀態,此研究結果可作為臨床照護胸腔重症病人脫離呼吸器使用之參考,以達成客觀評估一致性,降低病人呼吸器脫離延遲或失敗之傷害。

松科和羅漢松的質體基因組演化和親緣關係與裸子植物中乙酰輔酶A羧化酶基因的演化

為了解決APM Monaco的問題,作者陳章輝 這樣論述:

Plastid genomes (plastomes) serve as valuable and cost-effective genomic resources for plants and algae. More than 2,500 complete plastomes (as of December 2018) are now publicly available on GenBank, and they provide critical information on the evolution and phylogeny of plastid-bearing organisms.

In this dissertation, I will focus on the plastome evolution of non-flowering seed plants (gymnosperms). Gymnosperms comprise ca. 1,000 species in five groups, including cycads, ginkgo, gnetophytes, Pinaceae (conifers I), and cupressophytes (conifers II). Cupressophytes may be further divided into

five families: Cupressaceae, Taxaceae, Sciadopityaceae, Araucariaceae, and Podocarpaceae. Previous studies have highlighted that gymnosperm plastomes are highly variable. However, our understanding of the plastome evolution within gymnosperm families is incomplete because not all 12 families are equ

ally represented. In this study, I aimed to investigate (1) the plastome evolution and plastid phylogenomics of the two largest conifer families, Pinaceae and Podocarpaceae, and (2) the evolution of acetyl-CoA carboxylase (ACCase) genes in all five groups of gymnosperms.This dissertation has four ch

apters. In chapter one, I reviewed the available literature on gymnosperm plastids, plastome evolution, and ACCase. In chapter two, I reconstructed the complete plastid phylogenomics of Pinaceae by sequencing two Pinaceous genera, Pseudolarix and Tsuga. The intergeneric relationships among members o

f the Abietoideae subfamily were resolved with Cedrus as sister to the clade containing Pseudolarix-Tsuga and Abies-Keteleeria, which refutes previous phylogenetic studies. I also documented accD elongation in Pinaceae for the first time.In chapter three, I examined plastome evolution in the Podocar

paceae and expanded the number of available Podocarpaceae plastomes from 5 to 13. This addition enabled me to gain more insights into plastome evolution within the family. I found an exceptionally enlarged plastome in Lagarostrobos franklinii (Huon pine), a species endemic to Tasmania. Subsequent an

alyses revealed that the Lagarostrobos plastome is enriched with repetitive sequences, pseudogenes, and intergenic spacers that were not observed in other Podocarpaceae. In addition, plastid phylogenomic trees were also built to resolve problematic nodes in the Podocarpaceae phylogeny.In chapter fou

r, I investigated the evolutionary history of ACCase genes in the five gymnosperm groups. These genes are the key regulators of fatty acid biosynthesis, and most plants have both heteromeric and homomeric ACCases in plastids and cytosol, respectively. Heteromeric ACCase is composed of four subunits:

three nuclear-encoded accA–C and one plastid-encoded accD, while homomeric ACCase is only encoded by one nuclear ACC gene. This study uncovered that: (1) the ACCD subunit in all cupressophytes (except Sciadopitys) are elongated by lineage-specific tandem repeats, (2) Sciadopitys and gnetophytes hav

e functionally transferred their accD from the plastome to the nucleus, (3) Gnetum has two accDs in their nuclear genomes, and (4) one of Gnetum’s accD dually targets plastids and mitochondria, while the other copy only targets plastoglobuli, a microcompartment within the plastid. This is the first

study to report the presence of two accDs and their distinct targeting in any green plant.