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Panther Tank: The Panzerkampfwagen V in World War II

為了解決benz 250的問題，作者Doyle, David 這樣論述：

Featuring sloping armor and armed with a powerful 75 mm gun, Germany’s Panther tank was developed as a counter to the Soviet T-34. Entering production in 1943, the Panther was intended to become Germany’s standard medium tank. Although featuring potent armament and excellent armor, the Panther was h

ampered by disruptive production and drivetrain maintenance requirements. Produced in three variants, by three firms (MAN, Daimler-Benz, and MNH), the roughly 6,000 Panthers were used on the Eastern Front, in Italy, and from Normandy to Berlin. In this greatly expanded edition of the author’s earlie

r work, almost 250 photos are used to present the Panther from the factory to the battlefield in considerable detail.

benz 250進入發燒排行的影片

Metformin一種降血糖biguanide衍生物在離體及活體實驗中對抗血小板所引起的動脈栓塞所扮演的新角色

為了解決benz 250的問題，作者許嘉元 這樣論述：

第二型糖尿病(Type 2 diabetes, T2D)是一種內分泌異常所導致的疾病，近年來已成為全球第九大死亡原因。T2D的患者常伴隨著許多併發症例如：慢性腎臟疾病 (chronic kidney diseases, CKDs)、心血管疾病 (cardiovascular diseases, CVDs)、高血壓及神經退化性疾病，其中造成T2D患者死亡的原因主要為CKDs及CVDs。Metformin目前是治療T2D的一線臨床用藥，許多研究證明了metformin可以改善CVDs及CKDs；研究顯示血小板 (platelet) 在CVDs和腦血管疾病 (cerebrovascular dis

eases) 中扮演著關鍵角色，對於止血和動脈血栓的形成亦至關重要的作用。根據研究指出metformin (1 mM) 可以透過活化protein kinase A顯著降低棕櫚酸 (palmitic acid) 刺激的人臍靜脈內皮細胞 (human umbilical vein endothelial cell, HUVEC) 功能障礙和抑制藉由ADP (adenosine diphosphate) 刺激大鼠的血小板活化及粒線體功能障礙 (mitochondrial dysfunction)，顯見metformin對於生物體內的抗血小板效果卓越；因此metformin可能是治療動脈血栓形成的潛

在藥物。然而，在過去的文獻中僅有一篇研究證實了metformin在動物體內的抗血小板作用，因此在本研究中我們將首度使用人類血小板確認metformin在血小板活化中的分子機制，並進一步評估其在動物小鼠體內的有效性。研究結果顯示，metformin (1.2 mM~1.8 mM) 抑制膠原蛋白 (collagen, 1 μg/ml) 所刺激的血小板凝集作用 (aggregation) 但對於花生四烯酸 (arachidonic acid, AA; 60 μM)、U46619 (1 μM) 和凝血酶 (thrombin, 0.05 U/ml) 刺激的血小板活化沒有顯著影響。此外，metformin

(1.2 mM和1.8 mM) 顯著抑制由collagen所刺激的 ATP (adenosine triphosphate) 和鈣離子釋放、P-selectin的表現以及降低phospholipase C (PLC)γ2、protein kinase C (PKC)、mitogen-activated protein kinase (MAPK)、PI3K (phosphoinositide 3-kinase)、Akt及glycogen synthase kinase-3β (GSK3β) 的蛋白磷酸化，但對於vasodilator-stimulated phosphoprotein (VAS

P) 蛋白磷酸化並無影響。另外，在動物研究中metformin (250 mg/kg; i.p.) 明顯降低了急性肺栓塞 (acute pulmonary thromboembolism) 的死亡率且並不會造成出血的副作用產生。綜上所述，此研究結果證實了metformin可以作為有效抑制DM相關的血小板活化之新一類抗血小板藥物。

The Nextgen Guide to Car Collecting: Everything You Need to Know to Find, Buy, and Enjoy Collector Cars from the 1980s to Today

為了解決benz 250的問題，作者Yeager, Robert C. 這樣論述：

Robert C. Yeager has loved cars ever since his first ＄250 purchase at 16: a ＂raked,＂ baby-blue 1940 Ford coupe with headers. As an adult, he has owned a series of Alfa Romeos and currently enjoys a 1972 Mercedes-Benz 450 SL. Yeager spent his professional life as a writer for such outlets as Readers

Digest and Business Week and numerous newspapers including Chicago Tribune and New Orleans Times-Picayune. Yeager has also authored works of fiction and nonfiction for publishers like McGraw-Hill. For the past decade-plus he has covered classic cars for The New York Times. Yeager brings a seasoned c

ollector’s eye to the quickly growing ＂youngtimer＂ segment of the car hobby. He lives in California. McKeel Hagerty is a lifelong car lover and the Chief Executive Officer and driving force behind Hagerty, the world’s largest specialty automotive insurance provider. Under his leadership, Hagerty has

grown from a local insurance agency operating from his parents’ basement in Traverse City, Michigan, to a global market leader. Hagerty is the leading provider of specialty insurance protecting nearly 2 million classic and enthusiast vehicles. In 2017, McKeel broadened the company’s mission to savi

ng driving and growing the car community for future generations. To support those goals, the company has made a series of strategic partnerships, acquisitions, and expansions including the Hagerty Drivers Club (620,000 members), Hagerty Drivers Club magazine (600,000 circulation), Hagerty YouTube (1

.3 million subscribers), DriveShare (peer to peer rentals), the Greenwich Concours d’Elegance, and the industry gold standard for vehicle values, the Hagerty Valuation Tool. His favorite car is his first, which he drives to this day--a 1967 Porsche 911S that he found in pieces in snowbanks behind a

barn near his boyhood home and restored with the help of his dad.

語言、大腦與時間的相遇：漢語的觀點

為了解決benz 250的問題，作者理克 這樣論述：

時間被視為人類認知中至關重要的概念，時間標記則因語言而異。不過，大腦處理時間標記仍然不清楚。現今絕大多數的相關研究皆是針對印歐語系語言中的「時態」(tense)標記。本論文將要探討漢語中的「了」及「過」兩個「時貌」(grammatical aspect)標記和過去時間的關係，以及大腦如何處理「了」及「過」之時間標記。首先，本論文針對漢語中存在已久的「了」及「過」是否具有「時態」成分之辯論提供反證，並且主張「了」及「過」被用於表示過去時間是因為其時貌特徵。本論文將兩者時貌特徵與表示過去時間的關係分析成對應到不同語法的意義、功能：「了」對應到「時間先後關係」的認知功能，而「過」對應到「時間定位」

的認知功能。接著，本論文透過三個採用事件相關電位(event-related potential，稱ERP)技術的實驗測試/探討以上的分析。相關結果顯現了兩種質變上不同的效果進而支持上述分析：「了」在時間標記上的大腦處理誘發前腦及早期之負極性波幅(early frontal negativity)，而「過」則是誘發晚期之正極性波幅(P600)，此外，「了」及「過」在時間標記上的大腦處理方式與「了」及「過」在時貌上(非時間上)不同。最後，為使以上兩種大腦處理方式有廣義的且跨語言的功能解釋，本論文將漢語的時貌標記在時間上的大腦處理與印歐語系的時態標記在時間上的大腦處理方式做相互對比：前腦及早期之負

極性波幅(early frontal negativity)被視為反映「時間先後關係」的認知功能，晚期之正極性波幅(P600) 被視為反映「時間定位」的認知功能。總而言之，本論文採用以上分析及證據提供大腦如何處理時間標記之類型神經語言學模式。